nociceptive


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Related to nociceptive: allodynia, nociceptive reflex

no·ci·cep·tive

 (nō′sĭ-sĕp′tĭv)
adj.
1. Causing pain. Used of a stimulus.
2. Caused by or responding to a painful stimulus: a nociceptive spinal reflex.

[From nociceptor.]

nociceptive

(ˌnəʊsɪˈsɛptɪv)
adj
(Physiology) causing or reacting to pain
[C20: from Latin nocēre to injure + (re)ceptive]
ThesaurusAntonymsRelated WordsSynonymsLegend:
Adj.1.nociceptive - caused by or in response to pain; "a nociceptive spinal reflex"
sensitive - responsive to physical stimuli; "a mimosa's leaves are sensitive to touch"; "a sensitive voltmeter"; "sensitive skin"; "sensitive to light"
References in periodicals archive ?
In transmission, nociceptive transmission is modulated.
On the other hand, TMD can be considered a potential perpetuating factor for migraine because it acts as a constant nociceptive input that contributes to maintaining central sensitization and abnormal pain processes.
In pathological states, the relaying of noxious input by the nervous system is corrupted, resulting in abnormal nociceptive signaling and an aberration in pain responses (8).
The ablation resulted in hypersensitivity to acute nociceptive stimulation, exacerbated behavioral changes to chronic immobilization, and enhanced sodium absorption (3-5).
8] The venom-induced vasosensory reflex responses lasts longer than the other nociceptive agonists (capsaicin/anandamide/[alpha] [beta]Me-ATP)-induced responses.
New findings suggest that nociceptive sensory neurons (nociceptors) might regulate the development of immune responses.
Until recently, it was assumed that chronic pain worked much the same way as acute pain and was caused by ongoing nociceptive input in the periphery, but research has shown us that the central nervous system can play a large role in the modulation of nociception.
Nociceptive processes in craniofacial muscles are believed to play a role in the development and maintenance of TTH.
Nociceptive pain (resulting from inflammatory mediators) and neuropathic pain (secondary from injury and/or dysfunction of the somatosensory system) are frequent, as well as other sensory disturbances.
The objective of the technique was to shrink collagen fibrils by coagulating neural and inflammatory tissues of the disc, thus decreasing nociceptive input from the painful disc.
Dexamethasone, a steroid with anti-inflammatory properties block the nociceptive impulse transmission along the unmyelinated C fibres (11) and suppressing ectopic neuronal discharge.