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Both occur due to a complex cascade involving FGF-23 with reduction in phosphaturia and inhibition of 1-[alpha] hydroxylase.
Tenofovir causes Fanconi's syndrome characterised by proteinuria, phosphaturia, glycosuria (with normal blood glucose) and bicarbonate wasting.
Hypophosphatemia is the characteristic abnormality in these patients and is attributed to increased secretion of fibroblast growth factor 23 (FGF23), a phosphatonin secreted from dysplastic bone lesions, hence leading to phosphaturia (16).
Additionally, PTH causes phosphaturia, resulting in a low-normal or low serum phosphorus level.