functional recovery and BMIPP uptake after primary percutaneous transluminal coronary angioplasty in acute myocardial infarction.
Ischemic preconditioning attenuates postischemic
leukocyte - endothelial cell interactions role of nitric oxide and protein kinase C.
Studies also found that there was an increase in hypoxia of the renal medulla and in renal free-radical production through postischemic
oxidative stress after infusion of contrast medium (4).
Lee, "Fluoxetine affords robust neuroprotection in the postischemic
brain via its anti-inflammatory effect," Journal of Neuroscience Research, vol.
Another group of cardioprotective medications is based on their ability to improve the energy metabolism in a postischemic
heart [28, 29].
It is thought to occur due to small vessel rupture and/or postischemic
reperfusion injury [6, 7, 11].
This leads to increased cardiac workload with coronary microvascular spasm, consequent demand ischemia, and postischemic
angiogenesis also plays an important role in the long-term functional recovery .
treatment with anesthetic agents, such as remifentanil, sevoflurane, and morphine, has been suggested to induce cardioprotective effects in normal rats [10-12], while additional studies have shown that the cardioprotection of ischemic postconditioning and some anesthetic treatment may be blocked or abrogated in pathological conditions such as diabetes, hypercholesterolemia, and hyperglycemia [10-13].
Previous studies have revealed the role of P-selectin in IRI and sepsis model of AKI, and blocking P-selectin protects mice from AKI through attenuating neutrophil recruitment into kidney.[sup], Research also indicated that platelet P-selectin, but not endothelial, is critical for neutrophil-mediated acute postischemic
renal failure.[sup] Whether platelet-derived P-selectin is involved in the development of SAKI has not been determined.
Biochemical and histological investigation of famotidine effect on postischemic
reperfusion injury in the rat ovary.
Quercetin improves postischemic
recovery of heart function in doxorubicin-treated rats and prevents doxorubicin-induced matrix metalloproteinase-2 activation and apoptosis induction.