repolarization


Also found in: Medical, Wikipedia.

re·po·lar·i·za·tion

 (rē-pō′lər-ĭ-zā′shən)
n.
The restoration of a polarized state across a membrane, as in a muscle fiber following contraction.

repolarization

(ˌriːpəʊləraɪˈzeɪʃən) or

repolarisation

n
formal the act or process of polarizing again, a renewed polarization, the restoration of polarization
Translations

re·po·lar·i·za·tion

n. repolarización, restablecimiento de la polarización de una célula o de una fibra nerviosa o muscular después de su depolarización.
Mentioned in ?
References in periodicals archive ?
Long QT syndrome is a condition which affects repolarization of the heart after a heartbeat.
No significant change in repolarization was observed.
Patients with drug use that could affect ventricular repolarization were excluded from the study.
BK channels are widely expressed in the nervous system where they contribute to the repolarization of action potentials and afterhyperpolarization in neurons, beside regulating synaptic release of neurotransmitters.
Conclusion: Repolarization parameters increase in advanced-age pregnancies even though they remain in the normal range, which should lead to an investigation of whether this is a pathological condition.
The duration of the QT interval as measured by 12-lead electrocardiography is a measure of myocardial repolarization and is widely used to describe cardiac abnormalities, to determine the presence of cardiac toxicity and to evaluate drug safety.
The most common arrhythmia type was found to be supra ventricular tachycardia (SVT) 126 (44%), followed by premature ventricular contractions (PVCs) 80 (28%), early repolarization 34 (11.9%), Atrial Fibrillation (AF) 25 (8.7%), first degree heart block 15 (5.2%), Inappropriate sinus tachycardia (IST) 6 (2.1%).
Pseudoinfarct patterns on electrocardiogram mimicking myocardial infarction are seen in clinical conditions like pericarditis, myocarditis, ventricular aneurysm, takotsubo cardiomyopathy, early repolarization, and cardiac memory.
Myocardial damage results in altered electrical potential distributions and repolarization changes manifesting as prolonged Qt dispersion and electrophysiological changes in T-waves [3, 4].
The main purpose of this study was to investigate the effects of fibrosis on atrial excitability and repolarization. To achieve this goal, we used a proposed fibroblast model [12] coupled with the Courtemanche et al.
Some investigators contend that in patients with ventricular pacemakers the JTc, and not the QTc, interval is the more useful marker for assessing repolarization [10].
Electrocardiographic ventricular repolarization (QT interval) parameters have been used as predictors of increased risk of ventricular arrhythmias and sudden death in patients with SCI (Akbal et al., 2014; La Fountaine et al., 2010; Ravensbergen et al., 2012).