simian immunodeficiency virus


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simian immunodeficiency virus

n.
SIV.
References in periodicals archive ?
Simian immunodeficiency virus, infection, alcohol and host defense.
Mechanisms of minocycline-induced suppression of simian immunodeficiency virus encephalitis: inhibition of apoptosis signal-regulating kinase 1.
Molecular epidemiology of simian immunodeficiency virus SIVsm in U.
Chapters address lentivirus tropism and disease, macrophages in HIV-1 infection, intracellular mechanisms for lentiviral restriction, simian-HIV and simian immunodeficiency virus models of HIV disease, host and viral determinants of feline immunodeficiency virus cell tropism and pathogenicity, equine infectious anemia virus and its pathogenesis, the bovine lentivirus infections, and lentivirus coinfections and superinfections, among other topics.
Over the years, studies of simian immunodeficiency virus (SIV)-infected macaques and HIV-infected humans have provided substantial evidence that significant GI pathology results from progressive HIV-1 infection from the acute phase of the infection to advanced disease [2-9].
Along-term study of a wild population has found that chimpanzees naturally infected with simian immunodeficiency virus, or SIV, die early and their babies die within a year of birth.
It is closely related to gorilla simian immunodeficiency virus and shows no evidence of recombination with other HIV-1 lineages.
Ashley Haase and colleagues from the University of Minnesota have found that a few epithelial cells on the cervix of femal macaques are the first point of entry for simian immunodeficiency virus (SIV).
Ibaraki, Japan) has patented a method for highly efficient gene transfer into primate-derived embryonic stem (ES) cells has successfully been achieved by using a simian immunodeficiency virus vector (SIV) pseudotyped with VSV-G protein, which is a surface glycoprotein of vesicular stomatitis virus (VSV) The present invention provides simian immunodeficiency virus vectors for gene transfer to primate ES cells.
Data from simian modeling with simian immunodeficiency virus show that T-cell depletion in the gut occurs very rapidly in infection, and that early treatment can preserve some of these memory T cells, which may allow better immune-system control of HIV over a longer term.
The findings of the ten-year study to locate the source of HIV provide a crucial link between Simian Immunodeficiency Virus (SIVcpz), a virus similar to HIV that is well-known to affect chimpanzees, and the cause of human Aids.
Scientists believe hunters who ate infected animals contracted Simian Immunodeficiency Virus, which is very close to H.
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