transferrin


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Related to transferrin: Transferrin saturation, TIBC

trans·fer·rin

 (trăns-fĕr′ĭn)
n.
Any of a family of proteins that are found in the blood serum of humans and other vertebrates and that combine with and transport iron.

American Heritage® Dictionary of the English Language, Fifth Edition. Copyright © 2016 by Houghton Mifflin Harcourt Publishing Company. Published by Houghton Mifflin Harcourt Publishing Company. All rights reserved.

transferrin

(trænsˈfɜːrɪn)
n
(Biochemistry) biochem any of a group of blood glycoproteins that transport iron. Also called: beta globulin or siderophilin
[C20: from trans- + ferro- + -in]
Collins English Dictionary – Complete and Unabridged, 12th Edition 2014 © HarperCollins Publishers 1991, 1994, 1998, 2000, 2003, 2006, 2007, 2009, 2011, 2014

trans•fer•rin

(trænsˈfɛr ɪn)

n.
a plasma glycoprotein that transports dietary iron to the liver, spleen, and bone marrow.
[1947; trans- + Latin ferr(um) iron + -in1]
Random House Kernerman Webster's College Dictionary, © 2010 K Dictionaries Ltd. Copyright 2005, 1997, 1991 by Random House, Inc. All rights reserved.
ThesaurusAntonymsRelated WordsSynonymsLegend:
Noun1.transferrin - a globulin in blood plasma that carries iron
globulin - a family of proteins found in blood and milk and muscle and in plant seed
Based on WordNet 3.0, Farlex clipart collection. © 2003-2012 Princeton University, Farlex Inc.
Translations

trans·fer·rin

[MIM*190000]
n. transferrina, globulina beta en el plasma de la sangre que fija y transporta el hierro.
English-Spanish Medical Dictionary © Farlex 2012

transferrin

n transferrina
English-Spanish/Spanish-English Medical Dictionary Copyright © 2006 by The McGraw-Hill Companies, Inc. All rights reserved.
References in periodicals archive ?
Blood Tf binds to transferrin receptor 1 (TfR1) on endothelial cells lining blood capillaries and the binding complex is internalized into endosomes (Fig.
We compared circulating iron, total iron binding capacity, and percent transferrin saturation levels in apparently healthy wild and captive kori bustards.
(11) Increased ferritin alone is insufficient to warrant HFE genetic testing, but it should prompt testing for transferrin saturation.
The preserved material was used to determine the concentrations of transferrin, IgG, KIM-1, and uromodulin (uUMOD) in urine and NGAL, CysC, and uromodulin (sUMOD) in serum.
Using our records, we identified 23 patients who had low serum transferrin levels preoperatively.
Venous blood was drawn to evaluate hemoglobin levels, CRP, iron, ferritin, transferrin and transferrin saturation, glucose, and HbA1C.
Correspondingly, the soluble transferrin receptor-to-ferritin ratio was significantly decreased in groups 2, 3 and 4 (p<0.001).
Hepatic transferrin gene expression was down-regulated in G-II, in comparison with the control group, whereas in G-I and G-III, no detectable level was observed.
(4) More specialized tests to differentiate IDA from anemia of chronic disease (ACD) include hepcidin and transferrin receptor levels.
This disease is caused by mutations in transferrin receptor 2 (TFR2) gene [2] that codes for two main isoforms, namely, Tfr2 alpha (Tfr2[alpha]) and Tfr2 beta (Tfr2[beta]), that show moderate homology to the type 1 transferrin receptor (Tfr1) [3].
Transferrin saturation was determined by using the formula; iron/TIBC x100.