tricarboxylic

tri·car·box·yl·ic

 (trī′kär-bŏk-sĭl′ĭk)
adj.
Having three carboxyl groups.
References in periodicals archive ?
R], substrate carbon (or carbohydrate) retained in new biomass, or expended in maintenance or biosynthesis, respectively; TCA, tricarboxylic acid.
Most enzymes involved in intermediate metabolism have been shown to be acetylated, which include nearly all enzymes involved in glycolysis, glycogen metabolism, gluconeogenesis, the tricarboxylic acid cycle, fatty acid oxidation, the urea cycle and some enzymes involved in oxidative phosphorylation and amino acid metabolism [12,13].
Results from proteomics demonstrated that SXSM enhanced oxidative phosphorylation and tricarboxylic acid (TCA) cycle in ventricular myocardium to improve ATP generation.
In the latter mechanism, increased mitochondrial NADH causes inhibition of tricarboxylic acid cycle function, resulting in the accumulation of acetyl-CoA.
Moreover, PSII isnt the only component that is lacking in this genome: RuBisCo (ribulose-1,5 bisphosphate carboxylase-oxygenase that fixes CO2) and the tricarboxylic acid cycle (TCA) that usually are present in the cyanobacteria, are missing in UCYN-A.
The homogenate was centrifuged with equal parts of 10% tricarboxylic acid (TCA) at 5000 rpm for 10 min.
During respiration, proteins involved in the primary metabolic reactions in tricarboxylic acid cycle are catalyzed by copper and iron; thus, facilitate the electron transfer in ETC (Xu et al.
Citrate, 2-oxoglutarate and succinate are key intermediate products of tricarboxylic acid (TCA) cycle which involves not only the glucose aerobic oxidation but also the major pathways for fat and amino acid metabolisms.
Most of the cell energy is generated as adenosine triphosphate (ATP) within the mitochondria through two complementary processes: the tricarboxylic acid cycle (TCA) and oxidative phosphorylation (Barnes and Weitzman 1986; Dykens et al.
The degradation mechanisms of this polymer in aqueous media occur by simple hydrolytic scission into lactic acid, which is its monomeric form and that is eliminated from the body by incorporation into the tricarboxylic acid cycle.
824 As noted earlier DCA has been studied and shown to increase lactate clearance by speeding up the tricarboxylic acid (TCA) cycle.
The final products of both the pathways are molecules that can enter the Tricarboxylic acid cycle [8,9].