tricarboxylic acid cycle


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Related to tricarboxylic acid cycle: glycolysis, Electron transport chain

tricarboxylic acid cycle

tricarboxylic acid cycle

(traɪˌkɑːbɒkˈsɪlɪk)
n
(Biochemistry) biochem another name for Krebs cycleAbbreviation: TCA cycle

Krebs′ cy`cle


n.
the metabolic sequence of enzyme-driven reactions by which carbohydrates, proteins, and fatty acids produce carbon dioxide, water, and ATP.
[1940–45; after H. A. Krebs]
ThesaurusAntonymsRelated WordsSynonymsLegend:
Noun1.tricarboxylic acid cycle - in all plants and animals: a series of enzymatic reactions in mitochondria involving oxidative metabolism of acetyl compounds to produce high-energy phosphate compounds that are the source of cellular energy
metabolic process, metabolism - the organic processes (in a cell or organism) that are necessary for life
biological process, organic process - a process occurring in living organisms
oxidative phosphorylation - an enzymatic process in cell metabolism that synthesizes ATP from ADP
References in periodicals archive ?
(2007) Tricarboxylic acid cycle intermediate pool size: functional importance for oxidative metabolism in exercising human skeletal muscle.
Influence of environmental and nutritional factors on succinic acid production and enzymes of reverse tricarboxylic acid cycle from Enterococcus flavescens.
Succinate dehydrogenase consists of four protein subunits: SDHA to SDHD that are localized in the inner mitochondrial membrane and act at the interphase of the tricarboxylic acid cycle and electron transport chain.
OA detection with qualitative gas chromatography-mass spectrometry assays is problematic for 2 reasons: First, OA is a charged nitrogen species of high polarity and does not extract into organic solvents with high efficiency; second, OA often coelutes with the tricarboxylic acid cycle intermediate cis-aconitic acid on many gas chromatography columns.
Devlin, "The tricarboxylic acid cycle," in Textbook of Biochemistry with Clinical Correlations, pp.
This might be because the long-term high-fat diet led to energy metabolism disorders in rats, which affected gluconeogenesis and tricarboxylic acid cycle [29].
Sims [12] suggested that C[O.sub.2] could somehow drive the tricarboxylic acid cycle which drives energy for morphogenesis, which is an essential process in induction of Candida germ tubes [4, 12].
They suggested that ammonia may adversely affect the developing embryo by decreasing the concentration of a-ketoglutarate by converting it to glutamate, thereby reducing the flux through the tricarboxylic acid cycle (TCA) cycle and depleting ATP in embryonic cells and reducing the availability of ATP for embryonic cells during a stage of development.