spermatogenesis

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sper·mat·o·gen·e·sis

 (spər-măt′ə-jĕn′ĭ-sĭs, spûr′mə-tə-)
n.
Formation and development of spermatozoa by meiosis and spermiogenesis.

sper·mat′o·ge·net′ic (-jə-nĕt′ĭk), sper·mat′o·gen′ic (-jĕn′ĭk) adj.
American Heritage® Dictionary of the English Language, Fifth Edition. Copyright © 2016 by Houghton Mifflin Harcourt Publishing Company. Published by Houghton Mifflin Harcourt Publishing Company. All rights reserved.

spermatogenesis

(ˌspɜːmətəʊˈdʒɛnɪsɪs) or

spermatogeny

n
(Physiology) the formation and maturation of spermatozoa in the testis. See also spermatocyte1
spermatogenetic adj
Collins English Dictionary – Complete and Unabridged, 12th Edition 2014 © HarperCollins Publishers 1991, 1994, 1998, 2000, 2003, 2006, 2007, 2009, 2011, 2014

sper•mat•o•gen•e•sis

(spɜrˌmæt əˈdʒɛn ə sɪs)

n.
the origin and development of spermatozoa.
[1880–85]
Random House Kernerman Webster's College Dictionary, © 2010 K Dictionaries Ltd. Copyright 2005, 1997, 1991 by Random House, Inc. All rights reserved.
ThesaurusAntonymsRelated WordsSynonymsLegend:
Noun1.spermatogenesis - development of spermatozoa
gametogenesis - the development and maturation of sex cells through meiosis
Based on WordNet 3.0, Farlex clipart collection. © 2003-2012 Princeton University, Farlex Inc.
Translations
spermatogenèse
spermatogenesi

sper·ma·to·gen·e·sis

n. espermatogénesis, proceso de formación y desarrollo de espermatozoides.
English-Spanish Medical Dictionary © Farlex 2012
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References in periodicals archive
The increase in sperm concentration was due in part to the increase in testosterone and FSH levels in testicular tissue; since these two hormones were responsible for spermatocytogenesis and spermiogenesis in seminiferous tubules, while testosterone is responsible for epididymal function in maturation of sperms [100].
However, it is known that small increases in the amount of ROS in the testis can lead to exacerbation of apoptosis process not only during spermatocytogenesis, but also during spermiogenesis [6], which constitutes the later stages of spermatogenesis.
Our present study demonstrated that mice exposed to B[a]P at or greater than 50mg/kg/day for 30 days result in pathological changes such as decreasing spermatogonial cell proliferation and modulating germ cell apoptosis during spermatocytogenesis, the early stage of spermatogenesis.
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